By Arnold M. Katz (auth.), Wilson S. Colucci MD (eds.)
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Extra resources for Atlas of HEART FAILURE: Cardiac Function and Dysfunction
Q U depressed in patients with mitral regurgitation as reflected by a diminished ratio of pressure to volume at the end-systolic point, an estimate of ventricular elastance (sec Fig. 3-3). In chronic mitral regurgitation, the ventricle operates at a greater volume and, as with aortic regurgitation, ventricular size has proven to be predictive of impaired postoperative function. B, Echocardiogram with color Doppler depicting severe mitral regurgitation in a 70-year-old woman with a myxomatous mitral valve and a ruptured chordae.
Ventricular enlargement is a final common pathway in the heart with systolic impairment. c LV dia toli v lum , mUm1 c, Diastolic dysfunction also may produce heart failure, either primarily or in conjunction with systolic failure. Both intramyocyte (eg, alteration in excitation-contraction coupling and/ or cellular energetics) and extramyocyte phenomena (fibrosis, elevated afterload, and pericardia I constriction) contribute to diastolic dysfunction . The end-diastolic pressure-volume relationship (EDPVR) can be used to assess the passive properties of the ventricular chamber.
42. : Effect of positive inotropic agents on the relation between oxygen consumption and systolic pressurevolume area in canine left ventricle. Circ Res 1983,53:306-318. 43. : Velocity of contraction as a determinant of myocardial oxygen consumption. Am J Phvsiol 1965,209:919-927. 44. Gibbs CL, Gibson WR: Isoprenaline, propranolol and the energy output of rabbit cardiac muscle. Cardiovasc Res 1972, 6:508-515. 45. : Relation between oxygen consumption and pressure-volume area of in situ dog heart.
Atlas of HEART FAILURE: Cardiac Function and Dysfunction by Arnold M. Katz (auth.), Wilson S. Colucci MD (eds.)